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Although nicotine has a substantial impact on alterations in heart frequency, blood stress, and cardiac output caused by smoking, its involvement in atherothrombotic illness related to CS is still debated (56)

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-CBT-diol also protected the viability of neuro-2a cells by activating anti-apoptosis protein p-Akt and HAX-1, inhibiting pro-apoptosis protein caspase-3, and regulating the NF-B/VCAM-1/TNF- pathway to resist endothelial inflammation in murine brain-derived endothelial bEND5 cells