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Due to the incomplete understanding of the specific mechanisms of ferroptosis in AKI and the high mortality rate associated with AKI, exploring therapeutic targets for AKI from the perspective of ferroptosis may represent a novel direction with great potential for development

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Given that GABA is generally sleep-promoting (Gottesmann, 2002), this suggests a potential behavioral homeostatic feedback loop, with nitrogen stress accumulation during chronic sleep loss promoting the synthesis of Put-GABA, and thus sleep

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