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Its distinctive origin within mitochondrial DNA continues to make MOTS-c a valuable research peptide for exploring the mechanisms that regulate mitochondrial health and cellular resilience

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Fazekas et al., 2002) indicating that areas of NAWM, where a typical MS lesion will form in the future, exhibited significant abnormalities on T2 and magnetization transfer up to 2 years prior

The body takes what it needs and eliminates the rest, so there is no toxicity risk from these amounts