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While much has been learned about what up-regulates GCL expression, we remain largely in the dark about what down-regulates GCL, especially in disease states

Common examples include: These are designed to mimic or enhance the bodys own signaling molecules while extending their activity through modifications like PEGylation , fatty acid chains , or amino acid substitutions

This transcriptional regulator has been shown to play an important role in coordinating the oxidative stress response and its deletion in A

This theory is supported by the presence of inflammatory cells and immune system markers in the plaques of affected individuals